Rather bizzare considering the source. Obviously written by someone from the tobacco control industry.
Smoking Out E-cigarette Ad Claims | Truth In Advertising
Smoking Out E-cigarette Ad Claims | Truth In Advertising
Truth in Advertising pushing nothing but lies
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It would appear to be part of an orchestrated campaign to get some regulation in place before the ANTZ house of cards comes tumbling down.
The UKH report really lit a fire under them, eh?
The UKH report really lit a fire under them, eh?
May be the public health folks reason that the money brought in by cigarette taxes funds so much medical research that it's better for the public health to let smokers get sick and die and use their money to help other people with their illnesses.
Modern medicine works like this. The richest man in the world spends his entire fortune to create one pill. The pill is created. His fortune is gone. He takes the pill...and dies anyway.
Modern medicine works like this. The richest man in the world spends his entire fortune to create one pill. The pill is created. His fortune is gone. He takes the pill...and dies anyway.
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GO check out their drivel. Give them an earful.
Let's not leave ANYTHING like this unchallenged!
GO check out their drivel. Give them an earful.
Let's not leave ANYTHING like this unchallenged!
Those lying dirtbags smugly presume that nobody will follow the links. They claim "And several other key reports have not supported claims that e-cigarettes are safer. Those studies found that e-cigarettes contain harmful chemicals and can compromise immune systems in the lungs." The phrase "compromise immune systems in the lungs" links to: http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0116861 It's a stupid mouse study, which claims that "Cigarette smoke contains 1014 free radicals/puff [13] and causes oxidative damage, inflammation and apoptosis, which drive the pathogenesis of chronic obstructive pulmonary disease (COPD). More importantly, cigarette smoke exposure impairs innate and adaptive immune responses against bacteria and viruses, which predispose to COPD exacerbations, a major contributor to COPD-related morbidity, mortality and health care costs [14]." And reference [14] is this, http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1747235/pdf/v060p00925.pdf - WHICH FOUND THAT CURRENT CIGARETTE SMOKING WAS NOT A RISK FOR MORTALITY (current smoking HR 0.97 (0.63-1.48) univariate)! It just goes to show how their putrid movement operates by mindlessly parroting lies over and over again to deceive the public! And then those POS have the gall to pretend that e-cig vendors are lying!
I don't do Facebook, so feel free to post this info on it.
PS, the academic editor of that PLoS One mouse study was Dennis W. Metzger of Albany Medical College. Nice job of rubberstamping there, Dennis. It's great to see such enthusiasm for going above and beyond your minimal editorial duties such as merely checking out the links to see that they fit the claim. It's especially heartwarming to know that you should have known better than to endorse junk like that:
Inhibition of pulmonary antibacterial defense by interferon-gamma during recovery from influenza infection. K Sun, DW Metzger. Nat Med 2008 May;14(5):558-564. "We now report that pulmonary interferon-gamma (IFN-gamma) produced during T cell responses to influenza infection in mice inhibits initial bacterial clearance from the lung by alveolar macrophages. This suppression of phagocytosis correlates with lung IFN-gamma abundance, but not viral burden, and leads to enhanced susceptibility to secondary pneumococcal infection, which can be prevented by IFN-gamma neutralization after influenza infection. Direct inoculation of IFN-gamma can mimic influenza infection and downregulate the expression of the class A scavenger receptor MARCO on alveolar macrophages."
Inhibition of pulmonary antibacterial defense by interferon-gamma during recovery from influenza infection. - PubMed - NCBI
It means that a strong immune response against flu virus can increase the risk of pneumonia.
I don't do Facebook, so feel free to post this info on it.
PS, the academic editor of that PLoS One mouse study was Dennis W. Metzger of Albany Medical College. Nice job of rubberstamping there, Dennis. It's great to see such enthusiasm for going above and beyond your minimal editorial duties such as merely checking out the links to see that they fit the claim. It's especially heartwarming to know that you should have known better than to endorse junk like that:
Inhibition of pulmonary antibacterial defense by interferon-gamma during recovery from influenza infection. K Sun, DW Metzger. Nat Med 2008 May;14(5):558-564. "We now report that pulmonary interferon-gamma (IFN-gamma) produced during T cell responses to influenza infection in mice inhibits initial bacterial clearance from the lung by alveolar macrophages. This suppression of phagocytosis correlates with lung IFN-gamma abundance, but not viral burden, and leads to enhanced susceptibility to secondary pneumococcal infection, which can be prevented by IFN-gamma neutralization after influenza infection. Direct inoculation of IFN-gamma can mimic influenza infection and downregulate the expression of the class A scavenger receptor MARCO on alveolar macrophages."
Inhibition of pulmonary antibacterial defense by interferon-gamma during recovery from influenza infection. - PubMed - NCBI
It means that a strong immune response against flu virus can increase the risk of pneumonia.
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Perhaps Truth In Advertising, needs some truth in their articles.
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