So, just out of curiosity....
In a cart, you're getting:
1. Water
2. PG and/or VG
3. Nicotine (is there a chemical used to cut the pure stuff or is just diluted by water or glycerine)
4. Whatever chemicals/ingredients are used to make the flavorings
Is there anything else normally found in e-cig juice that doesn't fit in one of these 4 categories?
I guess what I'm asking is if you vape the unflavored carts, you're just getting water, pg/vg, and nicotine? No more, no less?
Alright...sticking with this thread sorry if it's a bit off topic.
The flavorings are sweet right... so that means they have to have some sort of sweetener in them. How many calories do you thing we're getting in our vapes?
Is this a stupid question? Just because we are inhaling instead of ingesting it's still going through our system. Right?
groundhog that's my favorite question of all time. I love you.
i wonder if they give you cavities...
o god. the anti-obesity drive. you've given the FDA ANOTHER angle...
"...and CHILDREN will inhale these things, they'll inhale them right along with their Big Macs and their Snickers Bars, and get FAT, and become overweight vape fiends with mailbox bombs and chinese disEASES, and free condoms, and they will spend their entire LIVES watching the mailbox for their FIX... we read about that stuff in a BIG VAPING forum..."
'Lynn, you're my hero!![]()
"Yes, it contains nearly unmeasurable trace amounts." "ZOMG! IT'S POISON! KILL THEM!" "No, it contains none." "We measured fifty thousand gallons and found a couple molecules. They said it didn't contain any! ZOMG! SUE THEM!"
'Lynn, you're my hero!![]()
Thank you as well, Magilla.
It's clear that I need to research, for my own edification, diacetyl inhalation with more depth. It's likely that, with a high toxicity rating, I'm already dead from a decade and a half of brewing (I tend to favor the rich ales that are enriched with naturally occuring diacetyl). And if I'm dead, then I'm obviously in hell - and that's a mean trick not to tell me.
Seriously, BPaulette, I think I'm diluting my own argument repeatedly in an attempt not to appear to be attacking you. You don't deserve my ire, and I'm not all that good at not being blunt, so that's probably where I'm falling down. So it's not my intent to step on your proverbial throat - just so you know.
what if you were vaping, drinking, and eating in the same space of say, an hour. Would that pose a higher risk?
Well, to be clear.... from what I've read, diacetyl (and apparently similar compounds now used as a substitute) is harmless when ingested. Its only harmful when inhaled, and only then when inhaled in large amounts. ...and they didn't figure this out until quite recently (the link between "popcorn lung" and the chemical wasn't made until 2000?). This hasn't affected a lot of people either - thus far, only workers in a popcorn factories, a couple of longtime popcorn freaks (who habitually ate at least 2 bags of popcorn a day for like 15 years), as well as a Blockbuster video employee who prepared several bags a night for the store.
Once they realized this, they immediately started using substitute chemicals. But the FDA is a bunch of dumbasses, as we know, and just within the last year, they've found that the substitute chemical does the same thing
...now, I'm no authority on this stuff, just been reading a lot - Google is your friend - and the link posted above by Volunteer Vapor is good general info as well
I would imagine that its like most things in life, if you inhale a small amount of this, you're fine. As Lynn pointed out, its a trace element in the air you breathe... it fills your kitchen when you make popcorn or reduce a wine sauce on the stove. But heavy vaping... where exactly does that fall in comparison to the amount inhaled nightly by a Blockbuster employee who makes several bags a night? I don't have a clue, because I have no data on volumes of either - no basis for comparison - and possibly nobody does
As example, butter flavor dosed at 0,3% in E juice. The amount of diacetyl in 100 ml of E.juice is 0,0045 grams. Assuming that a typical E cigarette contains an average of 2 ml of E juice, the amount of diacetyl assumed in a smoking session is...... 0,00009 grams or 0,09 ppm (part per million).
Always consider the dosage in use !
1506 AIRWAY EPITHELIAL TOXICITY OF THE FLAVORING AGENT, 2, 3-PENTANEDIONE.
[FONT=Times New Roman,Times New Roman]
[FONT=Times New Roman,Times New Roman]A. F. Hubbs[/FONT][FONT=Times New Roman,Times New Roman], A. E. Moseley, W. T. Goldsmith, M. C. Jackson, M. L. Kashon, [/FONT]
[FONT=Times New Roman,Times New Roman]L. A. Battelli, D. Schwegler-Berry, M. P. Goravanahally, D. Frazer, J. S. Fedan, [/FONT]
[FONT=Times New Roman,Times New Roman]K. Kreiss and V. Castranova. [/FONT][/FONT][FONT=Times New Roman,Times New Roman][FONT=Times New Roman,Times New Roman]NIOSH, Morgantown, WV. [/FONT][/FONT][FONT=Times New Roman,Times New Roman]
[/FONT][FONT=Times New Roman,Times New Roman]
[FONT=Times New Roman,Times New Roman]Workers producing microwave popcorn are at increased risk for severe, fixed airways obstruction. Human disease correlates with exposure to diacetyl (2,3-butanedione), a 4-[/FONT][/FONT][FONT=Times New Roman,Times New Roman][FONT=Times New Roman,Times New Roman]carbon, α[/FONT][/FONT][FONT=Times New Roman,Times New Roman][FONT=Times New Roman,Times New Roman]-diketone component of butter itself and many butter flavorings. In rats, acute diacetyl inhalation damages epithelium in nose, trachea and large intrapulmonary airways, with the greatest damage in nose, an injury distribution explained in part by the pharmacokinetics of inhaled diacetyl. A 5-[/FONT][/FONT][FONT=Times New Roman,Times New Roman][FONT=Times New Roman,Times New Roman]carbon α[/FONT][/FONT][FONT=Times New Roman,Times New Roman][FONT=Times New Roman,Times New Roman]-diketone, 2,3-pentanedione, is also used as a flavoring. The acute respiratory toxicity of 2,3-pentanedione, was investigated in this study because of structural similarities to diacetyl. Male, Sprague-Dawley rats inhaled 0, 118, 241, 318 or 354 ppm 2,3-pentanedione for 6 hr, were sacrificed the next day, and nose, trachea, and lung were assessed by histopathology. Airway epithelial changes included degeneration, apoptosis, necrosis and neutrophilic inflammation, with nasal epithelium being most affected. As exposure concentration increased, epithelial damage and inflammation increased in severity and extended deeper into the respiratory tract, with necrosuppurative tracheitis present in all rats inhaling 354 ppm. Physical examinations suggested delayed onset of toxicity. To investigate potential delayed toxicity, additional rats were exposed to 318 ppm, 2,3-pentanedione and sacrificed immediately (<2 hr) or 1 day (18 – 20 hr) after exposure. In the 1st nasal section (T1), minimal to mild, epithelial cell degeneration, apoptosis and individual cell necrosis observed immediately after exposure progressed with time post-exposure, developing into moderate to marked, multifocal and coalescent, necrosuppurative rhinitis the following day. These findings indicate that inhaled 2,3-pentanedione, similar to diacetyl, injures airway epithelium in rats, predominantly in nose, but also affects deeper airways. In addition, clinical and histopathologic toxicity are delayed after 2,3-pentanedione inhalation. [/FONT][/FONT][FONT=Times New Roman,Times New Roman][FONT=Times New Roman,Times New Roman]From: [/FONT][/FONT][FONT=Times New Roman,Times New Roman][FONT=Times New Roman,Times New Roman]The Toxicologist CD — An official Journal of the Society of Toxicology[/FONT][/FONT][FONT=Times New Roman,Times New Roman][FONT=Times New Roman,Times New Roman], [/FONT][/FONT][FONT=Times New Roman,Times New Roman]
[FONT=Times New Roman,Times New Roman]Volume 114, Number S-1, March 2010, page 319. [/FONT]
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