First, those are not Sx of nicotine withdrawal, nor do I think those are symptoms of nicotine overdose.
There are other drugs in your system. Were you a long time user of lorazepam or any other any benzodiazepine? The problem with benzo's is that they do NOT mix well with nicotine. Smokers are advised not to smoke while using this drug, as it seems to decrease the effectiveness of the drug. Therefore, they are prone to take more to reach the desired effect thus increasing their body's resistance to the drug. This is a cocktail for addiction.
IMO, stay on the sertraline. Anything less than 25mg won't do anything for you, so don't even bother. You may want to start with sertraline 25mg q.d. and see how that works for you. You might want to also think about adding a low dose of propanolol. Please consult your physician before you add, alter, or otherwise change a medication or course of therapy that he or she has prescribed for you.
Another thing...don't worry so much. Things will get better. Cheers.
no i dont have any drugs like benzos in my system especially before this happened. in fact ive never used ativan or xanax or valium or any of that before. ive been off zoloft for over a year and had just started getting mild anxiety about 2 wks or so before i started vaping so i restarted on 25mg of zoloft. 25 isnt enough and it never has been but i thought id try it. besides that my old Rx was actually expired over a year ago so there is a possibility it wasnt even giving 25mg. i should edit the first post because ive said a couple times since the first one that im sure it was cigarette withdrawal but not nicotine posioning. im on the zoloft 50mg and analogs now and this is my 3rd day and its made a huge improvement. i think its from coming off analogs that this has happened it started almost 3 days after i stopped them completely. that and the fact that i was getting ready to start going through the anxiety cycle again anyways.
so to clarify 3 weeks ago zero drugs in my system other than the 4000 chemicals in analogs
2 weeks ago 25mg zoloft only
last week e juice PG type
and this week analogs and zoloft 50mg so far ive only used 1 ativan and i doubt ill need more im feeling alot better.
im waiting on some PG to come in the mail so i can cut my 24mg juice to about 12mg that way i can assure my self im not getting to much and im going to smoke a couple analogs a day and slowwwwwwly ween myself from them.
and since my blood pressure and heart rate are (BP110/60-120/70 and HR60-70) excellent i would never take a beta blocker like propanolol nor would any doctor prescribe it for anxiety that i know of. using it for anxiety would not only be off lable it would be wayyyyy off label.
heres the side effects of beta blockers, beta blockers are dangerous. please folks dont try this sort of thing without your doctor explaining wth hes prescribing it in the first place.
i know some people are using this for stage freight, stupid and dangerous imo.
Adverse effects
Adverse drug reactions (ADRs) associated with the use of beta blockers include: nausea, ........, bronchospasm, dyspnea, cold extremities, exacerbation of Raynaud's syndrome, bradycardia, hypotension, heart failure, heart block, fatigue, dizziness, abnormal vision, decreased concentration, hallucinations, insomnia, nightmares, clinical depression, sexual dysfunction, erectile dysfunction and/or alteration of glucose and lipid metabolism. Mixed α1/β-antagonist therapy is also commonly associated with orthostatic hypotension. Carvedilol therapy is commonly associated with edema.[2]
Central nervous system (CNS) adverse effects (hallucinations, insomnia, nightmares, depression) are more common in agents with greater lipid solubility, which are able to cross the blood-brain barrier into the CNS. Similarly, CNS adverse effects are less common in agents with greater aqueous solubility (listed below).
Adverse effects associated with β2-adrenergic receptor antagonist activity (bronchospasm, peripheral vasoconstriction, alteration of glucose and lipid metabolism) are less common with β1-selective (often termed "cardioselective") agents, however receptor selectivity diminishes at higher doses. Beta blockade, especially of the beta-1 receptor at the macula densa inhibits renin release, thus decreasing the release of aldosterone. This causes hyponatremia and hyperkalemia.
A 2007 study revealed that diuretics and beta-blockers used for hypertension increase a patient's risk of developing diabetes while ACE inhibitors and Angiotensin II receptor antagonists (Angiotensin Receptor Blockers) actually decrease the risk of diabetes.[12] Clinical guidelines in Great Britain, but not in the United States, call for avoiding diuretics and beta-blockers as first-line treatment of hypertension due to the risk of diabetes.[13]
Beta blockers must not be used in the treatment of ......., amphetamine, or other alpha adrenergic stimulant overdose. The blockade of only beta receptors increases hypertension, reduces coronary blood flow, left ventricular function, and cardiac output and tissue perfusion by means of leaving the alpha adrenergic system stimulation unopposed. [14] The appropriate antihypertensive drugs to administer during hypertensive crisis resulting from stimulant abuse are vasodilators like nitroglycerin, diuretics like furosemide and alpha blockers like phentolamine. [15]
