I believe Stubby is correct, and is just stating what TropicalBob has been saying: there are many harm-reduction sources of nicotine, and perhaps ecigs are not enough for some people. I think people should perhaps be a little less sensitive towards statements that could be interpreted as a threat to ecigs. Knowledge of the real details of each of these harm-reducing methods is very much to our advantage, both on a personal level and in terms of the future of us being "allowed" to vape (I will likely continue regardless of whether I am allowed to or not).
It seems that the majority of nic we take in with vaping is orally absorbed, or throat/esophagus absorbed, rather than lung absorbed. It is being shown with these test results here and in other threads that vapers have elevated cotinine levels, but not in general greatly elevated nic levels. While the psychoactivity of cotinine is in some dispute, it seems to not be a active as nicotine.
I know for a fact that some people have highly metabolizing saliva, while others do not. Example: my father, when eating very thick starchy soup, will find that as he is eating his soup become thinner and more runny, while I personally do not have this effect. Why? Probably because his saliva is more active at breaking down starches than mine. More amylases in the saliva (although this is not prove, only a probable hypothesis).
So, perhaps you can see where I am going with this. My hypothesis: some people have more nic-oxidizing enzymes in their saliva, and so with them most of the nic is oxidized to cot orally. It could be that these are the people that show the lowest nic levels but higher cot levels, and perhaps vaping is not as effective for them. They have to chain vape high-nic juice to get the required fix.
Others may have lower oxiding enzyme levels orally, and so they are making less cot, and keeping more nic. So vaping at 6 or 8 mg is just fine for them, and they don't need high-nic binge vaping to kill the cravings.
So to test this, we need a calibrated oral ingestion of nicotine, not vaping. The lozenge comes in 2 mg and 4 mg strengths, and it is calibrated. Lets find out what the nic/cot levels are with lozenges, then we will know more about the oral actions of nicotine absorption. If both high-nic and low-nic users do this, it could tell us a lot. The lozenges take a little getting used to. You are supposed to just put it up in the corner of the mouth between cheek and gum, by the outside of an upper molar, and just leave it there. Minimally sucking on it, and no chewing it, which can cause niccups

.
What to you think? I am not interested in anyone stating this is a waste of time, and who cares anyway. Please keep those sentiments to yourself, as you simply do not understand scientific inquiry. Tell a nicotine researching biochemist that their investigations into nicotine nerve pain reduction methods are a waste of time, and let us know how that worked out for you. It is experiments like this that expand fields, and I say thank God there are people who after smoking for many years in blissful ignorance are actually craving more than nicotine. They are craving real data and knowledge. All good in my book.